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in comparison to radiograph, cardiomegaly and pulmonary vascular congestion are persistent findings with accompanying worsening diffuse interstitial opacities as well as increased areas of consolidation at the lung bases. observed findings may be due to pulmonary edema, likely coexisting with pneumonia with. small pleural effusions persist.
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mild pulmonary edema, small bilateral pleural effusions, and moderate cardiomegaly are little changed. the lung bases lobe better aerated, less likely to be pneumonia or even appreciable atelectasis. there is no pneumothorax. the transvenous right atrial lead is oriented more medially than usual. a lateral view would be helpful to determine that it is directed toward the anterior wall of the right atrium. transvenous right ventricular pacer defibrillator lead is nearly at the cardiac apex.
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no acute cardiopulmonary process.
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interval improvement in the right basilar opacity with stable appearance of right chest tube and right pleural effusion. no new consolidation.
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no pneumothorax.
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pa and lateral chest compared to and : the patient has had resection of two lung nodules. no nodules seen currently. asbestos-related pleural plaque, largely calcified, is extensive and unchanged. there is no new pleural effusion. heart size is normal. the mediastinal contour distorted by fat deposition is unchanged. there is no pneumothorax or pleural effusion. atherosclerotic calcification extending into the head and neck vessels and throughout the thoracic aorta is chronic, as is an upper thoracic vertebral compression fracture.
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new left upper lung field focal opacity which may reflect pneumonia in the correct clinical setting. follow up radiographs after treatment are recommend to ensure resolution of this finding.
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as compared to the previous radiograph, the diffuse and severe bilateral parenchymal opacities, reflecting pneumonia, have slightly increased in severity. moderate cardiomegaly with bilateral areas of atelectasis persist. no larger pleural effusions. no pneumothorax. unchanged appearance of the mediastinum.
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no acute cardiopulmonary process.
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no acute cardiopulmonary process.
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in comparison with the study of , there again are low lung volumes that accentuate the transverse diameter of the heart. bilateral atelectatic changes are seen at the bases, but no evidence of vascular congestion, pleural effusion, or acute focal pneumonia.
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compared to chest radiographs since , most recently at. <num> frontal chest radiographs taken in succession repositioning of the transesophageal feeding tube from the right main bronchus
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ap chest at compared to the severity of mild to moderate pulmonary edema is exaggerated by increasing large bilateral pleural effusions which are responsible for substantial bibasilar atelectasis, also worsening. there is no pneumothorax. severe cardiomegaly is a chronic problem, but never worse. mediastinal caliber suggests elevated central venous pressure or volume or both. et tube is in standard placement, an upper enteric drainage tube can be traced only as far as the mid esophagus, although it may go further, a right jugular line and ends in the mid to low svc. transvenous right atrial and right ventricular pacer leads are probably in standard positions, although the atrial lead is partially obscured.
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in comparison with the study of , the endotracheal and nasogastric tubes have been removed. layering pleural effusions are again seen bilaterally with volume loss predominantly in the left lower lung.
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no acute intrathoracic process
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grossly stable chest findings with left-sided loculated pneumothorax and trapped lung.
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in comparison to the previous radiograph from earlier today, mediastinal widening consistent with known hematoma appears unchanged. left lower lobe collapse has partially improved, and a small left pleural effusion is unchanged.
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no acute cardiopulmonary process.
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there no prior chest radiographs available for review. lungs are fully expanded and clear. heart size is top-normal; otherwise cardiomediastinal and hilar silhouettes and pleural surfaces are normal.
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findings concerning for sbo without definite signs of free air. basilar atelectasis is noted. consider ct to further assess.
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mild interval improvement in left upper lobe post-obstructive pneumonia/atelectasis.
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small right-sided pleural effusion. right basilar opacity may be due to atelectasis noting that infection would be difficult to exclude. ct would be more sensitive for the detection of metastatic disease.
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slight reduction in moderate left pneumothorax with no evidence of tension.
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patchy opacities in left upper lobe and both lung bases, findings concerning for infection in the correct clinical setting. probable mild pulmonary vascular congestion.
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no acute radiographic abnormality.
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no acute cardiopulmonary process.
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no evidence of acute cardiopulmonary disease.
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ap chest compared to through : mild to moderate pulmonary edema has increased since. moderate to large right pleural effusion, with a large chronic apical loculation has increased somewhat. heart is still severely enlarged. tracheostomy tube in place. large anterior chest mass is not appreciated on conventional chest radiographs.
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comparison to. an additional left-sided chest tube was inserted. there is a minimal decrease in extent of the left pleural fluid collection. the large subsequent atelectasis and biapical pleural lesion is stable. minimal atelectasis at the right lung basis.
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heart size is normal. mediastinum is normal. lungs are clear. there is no pleural effusion or pneumothorax.
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no acute cardiopulmonary process.
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no focal infiltrate.
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in comparison with the study , the patient is now upright rapid and supine. cardiac silhouette remains enlarged. the layering pleural effusions have now drop to the lower portion of the lungs. there is still a substantial right and relatively small left pleural effusion. the monitoring and support devices have all been removed.
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satisfactory position of right ij catheter without evidence of line related complications including pneumothorax.
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no acute cardiopulmonary abnormality.
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standard positions of the endotracheal and enteric tubes. the appearance of the chest remains otherwise unchanged compared to the exam from approximately min earlier.
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no previous images. cardiac silhouette is within normal limits and there is no evidence of acute pneumonia, vascular congestion, or pleural effusion.
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small left pneumothorax is unchanged. right pleural effusion is unchanged in size, with similar-appearing adjacent consolidation. left pleural effusion has increased, now moderate. right pneumothorax seen on prior cross-sectional study is not well appreciated
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no evidence of acute cardiopulmonary process.
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no convincing radiographic evidence of pneumonia is identified. small opacity at the left lung base is likely atelectasis.
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focal opacity in the right infrahilar region which could represent atelectasis or early focus of pneumonia. new interstitial opacities consistent with mild pulmonary edema. small bilateral pleural effusions.
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support lines and tubes are unchanged in position. cardiomediastinal silhouette is within normal limits. there are no focal consolidations, pleural effusion, or pulmonary edema. there are no pneumothoraces.
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unchanged bibasilar atelectasis and small left pleural effusion.
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in comparison with the study of , there is little change and no evidence of acute cardiopulmonary disease. no pneumonia, vascular congestion, or pleural effusion.
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ill defined left lower lobe opacity, possibly infection versus aspiration.
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large left pleural abnormality which increased between and has remained essentially stable. previous enlargement of the cardiac silhouette, perhaps due to pericardial effusion, has decreased. the extent of atelectasis in the left upper lobe is stable. the right lung is essentially clear and there is no right pleural effusion.
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ap chest compared to : cardiomegaly is severe. widening of the upper mediastinum could be due to mediastinal fat deposition and vascular engorgement. pulmonary vasculature is normal, and there is no edema or appreciable pleural effusion. no pneumothorax.
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no acute intrathoracic process.
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no acute cardiopulmonary process.
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no bulky lymphadenopathy to suggest sarcoidosis. findings concerning for ascending aortic aneurysm. recommend dedicated ct for further evaluation.
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findings consistent with emphysema, but no definite acute process.
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no acute cardiopulmonary process.
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multifocal patchy ill-defined opacities with a cavitary lesion noted in the superior segment of the left lower lobe. findings are compatible with multifocal pneumonia, and are highly concerning for tuberculosis.
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normal heart lungs hila mediastinum and pleural surfaces.
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mild pulmonary vascular congestion, similar to that seen on the prior study, without focal consolidation.
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worsening right multifocal pneumonia.
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no acute cardiopulmonary process.
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no acute cardiopulmonary abnormality.
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progression of bibasilar consolidations, in particular on the right, concerning for pneumonia.
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no acute cardiopulmonary process.
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bibasilar atelectasis. no focal consolidation.
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no acute intrathoracic process.
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patchy opacities throughout the right lung and a moderate right pleural effusion could represent pneumonia in the correct clinical setting. lines and tubes in appropriate position.
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normal chest radiographic examination.
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findings consistent with mild congestive heart failure. no frank pulmonary edema.
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continued volume loss at the right lung base with stable support devices. interval removal of right internal jugular central venous line.
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no radiographic findings to suggest sequela of asbestos exposure. decrease in heart size and resolution of pulmonary edema suggests improvement in congestive heart failure.
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no acute pulmonary process identified. in particular, no pleural effusion detected. nodular density at the right lung base corresponds the calcified granuloma seen on the ct scan.
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no acute cardiopulmonary process. re- demonstration of a <num> mm left pulmonary nodule.
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no acute cardiopulmonary abnormality. mild hyperinflation.
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compared to chest radiographs since , most recently. thoracic aorta is generally large, but not focally aneurysmal. heart is normal size. pulmonary and mediastinal veins are not distended. no pleural effusion. aside from linear scar at the left base lungs are well expanded and clear.
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no evidence of acute cardiopulmonary process.
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no acute cardiopulmonary process.
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no acute intrathoracic abnormality.
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in comparison with the study of , there is little change. monitoring and support devices remain in place. diffuse bilateral pulmonary opacifications, much of which has a nodular appearance, is again seen bilaterally, more prominent on the right.
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left lower lobe is collapsed with left mediastinal shift. right lung base atelectasis is less severe compared to left. superimposed pneumonia is possible in correct clinical setting. large left pleural effusions increased. moderate right pleural effusion is stable. pulmonary vascular congestion is reflective of cardiac decompensation.
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no radiographic evidence for acute cardiopulmonary process. rightward trachea deviation.
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et tube in good position. right picc catheter tip now in the distal svc. no significant change from prior radiograph.
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no acute intrathoracic process.
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small bilateral pleural effusions increased since. pulmonary hila are chronically enlarged. mediastinal caliber has decreased since when the chest ct showed mediastinal adenopathy in the prevascular station. very mild interstitial pulmonary abnormality could be early edema. if symptoms persist after cardioversion, i would recommend chest ct scanning to look for interstitial lung disease.
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no acute cardiopulmonary process seen.
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lung volumes are low. there are patchy bibasilar opacities suggestive of atelectasis. overall cardiac and mediastinal contours are stably enlarged. crowding of the perihilar vasculature is seen in the setting of low lung volumes. no pulmonary edema. no pleural effusions. no pneumothorax. hardware is seen overlying the mid-to-lower thoracic spine.
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no acute cardiopulmonary process.
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hypoinflated lungs with no acute cardiopulmonary findings.
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no acute cardiopulmonary process.
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low lung volumes with likely left basilar atelectasis.
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as compared to the previous examination, the lung volume on the right has further decreased. the right hilar mass is now surrounded by an upper lobe predominant parenchymal opacity with air bronchograms. this opacity has substantially increased since the previous examination. the size of the cardiac silhouette remains unchanged. normal appearance of the left lung.
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no acute cardiopulmonary process.
MIMIC-CXR-JPG/2.0.0/files/p13546197/s56496944/45476c61-73e7a2de-049a2913-d1ebd05e-bfaf9dc9.jpg
no evidence of pneumonia.
MIMIC-CXR-JPG/2.0.0/files/p18297984/s54567350/6c3fa992-f48af6f3-04765e90-98f55f97-3ece8b17.jpg
no evidence of pneumonia.
MIMIC-CXR-JPG/2.0.0/files/p17470895/s53047468/5b98c5b2-90354645-df6138b7-139ed812-814bb625.jpg
no acute intrathoracic process
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as compared to the previous radiograph, no relevant change is seen. normal structure and transparency of the lung parenchyma. no pneumonia, no pulmonary edema, no pleural effusions. normal hilar and mediastinal contours. normal size and shape of the cardiac silhouette.
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ng tube in the stomach.
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no acute cardiopulmonary process.
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diffuse basilar bronchiectasis, right greater than left, unchanged from prior. increased right lower lobe opacity raises concern for acute infection
MIMIC-CXR-JPG/2.0.0/files/p13028893/s51810673/61b2a977-70993818-c3012314-c5ca38f8-d5641395.jpg
no acute pneumonia.
MIMIC-CXR-JPG/2.0.0/files/p19001200/s53665476/c8caed40-906a864c-780beee8-0939b2d8-9871eb16.jpg
no acute cardiopulmonary process.
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no definite evidence of pneumonia. unchanged mild elevation of the right hemidiaphragm.
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compared to prior chest radiographs since , most recently. small left pleural effusion is chronic. mild to moderate cardiomegaly stable. lungs clear.
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no evidence of acute cardiopulmonary abnormalities.