question: Why does our ear(s) randomly ring for a short period of time and slowly recede?
comment: Your ears are actually just a lot of small hairs that detect noises around you. Some detect very high, some low, and many varying from loud to really quiet. When your ears are ringing, it usually means they are retuning. This might mean a difference in air pressure outside, or because you went from a silent space to a loud area or even just because biology with wax buildup and the ears ability to catch surrounding noises. After a little while, your hearing will be tuned for your current conditions, and the ringing will be tuned out or ignored effectively. (Edit) Touching on some more reasons for ears to ring: *Note: Ringing usually lasts longer here* * Tinnitus * Ear Infections * Inner Ear changes * Blood Pressure * Tumors / Trauma in the Head or Neck
passages: Other less common symptoms (all less than 15%) of cholesteatoma may include pain, balance disruption, tinnitus, earache, headaches and bleeding from the ear. There can also be facial nerve weakness. Balance symptoms in the presence of a cholesteatoma raise the possibility that the cholesteatoma is eroding the balance organs in the inner ear.
  
  Doctors' initial inspections may only reveal an ear canal full of discharge. Until the doctor has cleaned the ear and inspected the entire tympanic membrane, cholesteatoma cannot be diagnosed.
   Section::::Symptoms.
  
  The symptoms of endolymphatic hydrops include the feeling of pressure or fullness in the ears, hearing loss, tinnitus (ringing in the ears) and balance problems. Individuals who have Meniere’s disease have a degree of endolymphatic hydrops that is strong enough to trigger the symptoms of this disease, but individuals with endolymphatic hydrops do not always progress to Meniere’s disease.
  
  Section::::Causes.
   Section::::Sudden sensorineural hearing loss.:Presentation.
  
  Many people notice that they have SSHL when they wake up in the morning. Others first notice it when they try to use the deafened ear, such as when they use a phone. Still others notice a loud, alarming "pop" just before their hearing disappears. People with sudden deafness often become dizzy, have ringing in their ears (tinnitus), or both.
  
  Section::::Sudden sensorineural hearing loss.:Diagnosis.
   BULLET::::- Brain fog
  
  BULLET::::- Fatigue
  
  BULLET::::- Headache/migraine
  
  BULLET::::- Tinnitus – high pitched ringing in the ear
  
  Section::::Symptoms.:Symptoms in detail.
   Other symptoms can include photophobia, excessive lacrimation, fever, and reduced corneal sensitivity. The syndrome is classically caused by the spread of an infection into the petrous apex of the temporal bone.
  
  Section::::Diagnosis.
  
  The constellation of symptoms was first described as a consequence of severe, advanced ear infection which has spread to a central portion of the temporal bone of the skull. This type of presentation was common prior to development of antibiotic treatments, and is now a rare complication.
   Section::::Treatment.:Complications due to surgery.:Tinnitus.
  
  Most patients present with tinnitus before treatment, and also have it after treatment. About one in 5 patients without tinnitus acquire it, and for about 2 in 5 with tinnitus it resolves or decreases.
  
  Section::::Treatment.:Complications due to surgery.:Hearing loss.
   Ménière's disease was first identified in the early 1800s by Prosper Ménière. It affects between 0.3 and 1.9 per 1,000 people. It most often starts in people 40 to 60 years old. Females are more commonly affected than males. After 5 to 15 years of symptoms, the episodes of the world spinning generally stop and the person is left with mild loss of balance, moderately poor hearing in the affected ear, and ringing in their ear.
  
  Section::::Signs and symptoms.
   Temporomandibular disorders were described as early as ancient Egypt. An older name for the condition is "Costen's syndrome", eponymously referring to James B. Costen. Costen was an otolaryngologist, and although he was not the first physician to describe TMD, he wrote extensively on the topic, starting in 1934, and was the first to approach the disorder in an integrated and systematic way. Costen hypothesized that malocclusion caused TMD, and placed emphasis on ear symptoms, such as tinnitus, otaglia, impaired hearing, and even dizziness. Specifically, Costen believed that the cause of TMD was mandibular over-closure, recommending a treatment revolving around building up the bite. The eponym "Costen syndrome" became commonly used shortly after his initial work, but in modern times it has been dropped, partially because occlusal factors are now thought to play little, if any, role in the development of TMD, and also because ear problems are now thought to be less associated with TMD. Other historically important terms that were used for TMD include "TMJ disease" or "TMJ syndrome", which are now rarely used.
   Cholesteatoma is diagnosed by a medical doctor by physical examination of the ear. A CT scan may help to rule out other, often more serious causes for the patient's clinical presentation. Non-ionizing radiation imaging techniques (MRI) may be suitable to replace a CT scan, if determined necessary by your physician.
  
  Section::::Treatment.
  
  Cholesteatoma is a persistent disease. Once the diagnosis of cholesteatoma is made in a patient who can tolerate a general anesthetic, the standard treatment is to surgically remove the growth.
   BULLET::::- Pulsatile tinnitus is yet another of the typical symptoms of SCDS and is caused by the gap in the dehiscent bone allowing the normal pulse-related pressure changes within the cranial cavity to enter the inner ear abnormally. These pressure change thus affect the sound of the tinnitus which will be perceived as containing a pulse-synchronized "wave" or "blip" which patients describe as a "swooshing" sound or as being like the chirrup of a cricket or grasshopper.
   Limited data is available for the long-term prognosis of ring chromosome 20 syndrome since only over 60 patients with this syndrome have been reported in published literature. Optimal control of seizures appears to be the determining factor, but early diagnosis and a comprehensive management plan with multidisciplinary support is also thought be to be important.
  
  Section::::Research.
   Superior semicircular canal dehiscence syndrome is a set of hearing and balance symptoms, related to a common but rarely diagnosed medical condition of the inner ear, known as "superior canal dehiscence". The symptoms are caused by a thinning or complete absence of the part of the temporal bone overlying the superior semicircular canal of the vestibular system. There is evidence that this rare defect, or susceptibility, is congenital. There are also numerous cases of symptoms arising after physical trauma to the head. It was first described in 1998 by Lloyd B. Minor of Johns Hopkins University in Baltimore.
  
  Section::::Symptoms.
   Most patients with otosclerosis notice tinnitus (head noise) to some degree. The amount of tinnitus is not necessarily related to the degree or type of hearing impairment. Tinnitus develops due to irritation of the delicate nerve endings in the inner ear. Since the nerve carries sound, this irritation is manifested as ringing, roaring or buzzing. It is usually worse when the patient is fatigued, nervous or in a quiet environment.
  
  Section::::Causes.
   Use of certain medications with a ruptured tympanic membrane can cause tinnitus, vertigo, dizziness and hearing loss in some cases.
  
  Section::::Prognosis.
  
  Otitis externa responds well to treatment, but complications may occur if it is not treated. Individuals with underlying diabetes, disorders of the immune system, or history of radiation therapy to the base of the skull are more likely to develop complications, including malignant otitis externa. In these individuals, rapid examination by an otolaryngologist (ear, nose, and throat physician) is very important.
  
  BULLET::::- Chronic otitis externa
  
  BULLET::::- Spread of infection to other areas of the body
  
  BULLET::::- Necrotizing external otitis
   BULLET::::- In some children without “classic” holoprosencephaly, microforms of holoprosencephaly may be noted on MRI, including missing olfactory tracts and bulbs and absent or hypoplastic corpus callosum.
  
  BULLET::::- Strabismus as well as nystagmus have both been reported in infants and children with ring 18.
  
  BULLET::::- Hearing loss has been reported and may be related to ear canal atresia or stenosis.
  
  BULLET::::- Umbilical and inguinal hernias have been reported in a small number of people with ring 18.
   Section::::Signs and symptoms.
   Pain is the most common symptom, followed by either sensorineural or conductive hearing loss, tinnitus or drainage (discharge). A mass lesion may be present, but it is often slow growing.
  
  Section::::Diagnosis.
  
  Section::::Diagnosis.:Imaging.
  
  Imaging studies are used to define the extent of the tumor and to exclude direct extension from the parotid gland or nasopharynx. The imaging findings are usually non-specific, and cannot give a specific diagnosis.
  
  Section::::Diagnosis.:Pathology.
   BULLET::::2. Episodic, characteristic definitive spells of vertigo lasting 20 minutes to 24 hours with no unconsciousness, vestibular nystagmus always present.
  
  BULLET::::3. Tinnitus (ringing in the ears, from mild to severe) Often the tinnitus is accompanied by ear pain and a feeling of fullness in the affected ear. Usually, the tinnitus is more severe before a spell of vertigo and lessens after the vertigo attack.
  
  BULLET::::4. Attacks are characterized by periods of remission and exacerbation.
   Section::::Characteristics.
  
  Attacks of skin redness and burning sensation or pain in one or both external ears are the only common symptom. Pain is often most pronounced at the ear lobe, and sometimes radiates to the jawbone and cheek. The pain is normally mild, but has occasionally been described as severe. The attacks can last seconds or hours, with 30 minutes to an hour being typical. Most patients have daily attacks, ranging from 20 a day to a few a year.
  
  Section::::Causes.
   Investigations such as magnetic resonance imaging or CT scanning and electroencephalograms (EEGs) may be worthwhile, but will rarely show any serious pathology. It is believed that because this kind of phenomenon is usually heterogenous in causation, a wide variety of factors need to be considered, which could give possible explanation for why MES is seen as under diagnosed. Some of these factors may include significant trauma to the head or any side-effects from substances such as antidepressants, marijuana, alcohol, procaine, or general anesthesia.
  
  Section::::Treatment.
   The ECG findings in atrial septal defect vary with the type of defect the individual has. Individuals with atrial septal defects may have a prolonged PR interval (a first-degree heart block). The prolongation of the PR interval is probably due to the enlargement of the atria common in ASDs and the increased distance due to the defect itself. Both of these can cause an increased distance of internodal conduction from the SA node to the AV node.
   Section::::Diagnosis.:Severity.
  
  The condition is often rated on a scale from "slight" to "catastrophic" according to the effects it has, such as interference with sleep, quiet activities and normal daily activities. In an extreme case, a man committed suicide after being told there was no cure.
   Ménière's disease usually starts confined to one ear; it appears that it extends to both ears in about 30% of cases.
  
  People may start out with only one symptom, but in MD all three appear with time. Hearing loss usually fluctuates in the beginning stages and becomes more permanent in later stages. MD has a course of 5–15 years, and people generally end up with mild disequilibrium, tinnitus, and moderate hearing loss in one ear.
  
  Section::::Epidemiology.
   Section::::Thalamocortical circuits.:Resonant columns.
   The diagnosis of TR may be suspected if the typical murmur of TR is heard. Severe TR may be suspected if right ventricular enlargement is seen on chest x-ray, and other causes of this enlargement are ruled out. 
  
  Definitive diagnosis is made by echocardiogram, which is capable of measuring both the presence and the severity of the TR, as well as right ventricular dimensions and systolic pressures.
  
  Section::::Management.
  
  Section::::Management.:Medical.
  
  Generally, medical rather than surgical treatment of TR is recommended if the cause is right ventricular dilatation or left-sided heart failure.
  
  Section::::Management.:Surgical.
  
raw_presuppositions: Ear(s) randomly ring for a short period of time.
raw_corrections: Ears ring to readjust under certain types of change in the environment; this can cause a ringing sensation.