question: Why do people feel random pains in their body’s at random points in time? Someone mentions a scientific perspective but I need facts
comment: Your system shares neural pathways. Think of your nervous system like a multi lane highway. Some lanes for pain some for emotion. When you are very stressed you have a lot of traffic on the emotional lanes, some of it will change lanes into the pain lanes and register as pain. This can be a quick stabbing pain in random locations or ongoing chronic signals of pain coming from unresolved protracted internal or external stressors.
passages: Spontaneous pain or allodynia (pain resulting from a stimulus which would not normally provoke pain, such as a light touch of the skin) is not limited to the territory of a single peripheral nerve and is disproportionate to the inciting event.
  
  BULLET::::1. There is a history of edema, skin blood flow abnormality, or abnormal sweating in the region of the pain since the inciting event.
  
  BULLET::::2. No other conditions can account for the degree of pain and dysfunction.
   BULLET::::1. The presence of an initiating noxious event or a cause of immobilization
  
  BULLET::::2. Continuing pain, allodynia (perception of pain from a nonpainful stimulus), or hyperalgesia (an exaggerated sense of pain) disproportionate to the inciting event
  
  BULLET::::3. Evidence at some time of edema, changes in skin blood flow, or abnormal sudomotor activity in the area of pain
  
  BULLET::::4. The diagnosis is excluded by the existence of any condition that would otherwise account for the degree of pain and dysfunction.
  
  According to the IASP, CRPS II (causalgia) is diagnosed as follows:
   BULLET::::1. The presence of continuing pain, allodynia, or hyperalgesia after a nerve injury, not necessarily limited to the distribution of the injured nerve
  
  BULLET::::2. Evidence at some time of edema, changes in skin blood flow, or abnormal sudomotor activity in the region of pain
  
  BULLET::::3. The diagnosis is excluded by the existence of any condition that would otherwise account for the degree of pain and dysfunction.
   Teams root of the upper limbs
  
  The first sign of disease, sometimes before a few years, the occurrence of radicular syndrome are pain in the neck and shoulder area. Usually, the pain occurs as a consequence of a cold or hypothermia, sometimes calls it load, poor working posture or sleep, sudden movement of the head. Teams root localized mostly within the three lower cervical roots C5, C6, C7.
  
  Section::::Pain and pathologies.:Symptoms.
  
  BULLET::::- Forced, reflexive position of the spine (the slope and a slight twist in the opposite direction has fallen Root)
  
  BULLET::::- Paraspinal muscle contracture
  
  BULLET::::- Reduction of cervical lordosis
   BULLET::::- Pain associated with temporomandibular joint disorder and myofascial pain also often occurs in the same region as pericoronitis. They are easily missed diagnoses in the presence of mild and chronic pericoronitis, and the latter may not be contributing greatly to the individual's pain (see table).
   Events leading up to the injury and/or illness
  
  Section::::Interviewing / Physical exam.:Orthopaedic Assessment.
  
  CLORIDE FPP
  
  Character: sharp or dull pain
  
  Location: region (joint) of origin
  
  Onset: sudden vs. gradual
  
  Radiation:
  
  Intensity: how severe (scale 1-10), impact on ADLs (activities of daily living), is it getting better, worse or staying the same?
  
  Duration: acute vs. chronic
  
  Events associated: falls, morning stiffness, swelling, redness, joint clicking or locking, muscle cramps, muscle wasting, movement limitation, weakness, numbness or tingling, fever, chills, trauma (mechanism of injury), occupation activities, sports, repetitive movements
  
  Frequency: intermittent vs. constant, have you ever had this pain before?
   Pain is present in about 78% of cases. Slight pain is present in the earliest stage of ainhum, caused by pressure on the underlying nerves. Fracture of the phalanx or chronic sepsis is accompanied with severe pain.
  
  Section::::Cause.
   Precordial catch syndrome
  
  Precordial catch syndrome (PCS) is a non-serious condition in which there are sharp stabbing pains in the chest. These typically get worse with breathing in and occur within a small area. Spells of pain usually last less than a few minutes. Typically it begins at rest and other symptoms are absent. Concerns about the condition may result in anxiety.
   In addition to peripheral mechanisms, spinal mechanisms are thought to have an influencing role in phantom pains. Peripheral nerve injury can lead to the degeneration of C fibers in the dorsal horn of the spinal cord, and terminating A fibers may subsequently branch into the same lamina. If this occurs, A fiber inputs could be reported as noxious stimuli. Substance P, involved in the transmission of pain signals, is usually expressed by Aδ and C fibers, but following peripheral nerve damage, substance P is expressed by Aβ fibers. This leads to hyperexcitability of the spinal cord, which usually occurs only in the presence of noxious stimuli. Because patients with complete spinal cord injury have experienced phantom pains, there must be an underlying central mechanism responsible for the generation of phantom pains.
   Complex regional pain syndrome is uncommon, and its cause isn't clearly understood. Treatment is most effective when started early. In such cases, improvement and even remission are possible. CRPS typically develops after an injury, surgery, heart attack, or stroke. The pain is out of proportion to the initial event.
   Section::::Diagnosis.:Thermography.
   There are many painful conditions that can cause intractable pain disease. Not every patient with these conditions will develop intractable pain, but the following conditions are known to cause Intractable Pain in some patients:
  
  BULLET::::- Failed Back Syndrome
  
  BULLET::::- Scoliosis, Kyphosis, kyphoscoliosis
  
  BULLET::::- Degenerative Disk Disease
  
  BULLET::::- Rheumatoid Arthritis
  
  BULLET::::- Central Pain Syndrome (CPS)
  
  BULLET::::- Migraine
  
  BULLET::::- Ehlers-Danlos Syndromes
  
  Section::::Notable Figures.
  
  Although not recognized during their lifetimes, John F Kennedy and Howard Hughes are both believe to have suffered from Intractable Pain.
  
  Section::::References.
  
  BULLET::::- The intractable pain patient's handbook for survival, Forrest tenant, md, phd 2007, https://docs.google.com/viewer?a=v&pid=sites&srcid=ZGVmYXVsdGRvbWFpbnx0aGVjaHJvbmljcGFpbnxneDo2MTBiNGY1MGFhZTg3MzM1
   Section::::Research.
   Symptoms may change over time, and they can vary from person to person. Symptoms can even change numerous times in a single day.
  
  The pain of CRPS is continuous although varies in severity. It is widely recognized that it can be heightened by emotional or physical stress.
   Section::::Pathophysiology.
   Section::::Syndromes.
  
  BULLET::::- Upper limb
  
  BULLET::::- Lower limb, abdomen and pelvis
  
  Section::::Signs/symptoms.
  
  Tingling, numbness, and/ or a burning sensation in the area of the body affected by the corresponding nerve. These experiences may occur directly following insult or may occur several hours or even days afterwards. Pain is less common than tingling or numbness as a symptom of nerve entrapment, although a burning sensation, if it occurs, may (subjectively) be classified as pain.
  
  Section::::Causes.
   The prevalence of phantom pain in upper limb amputees is nearly 82%, and in lower limb amputees is 54%. One study found that eight days after amputation, 72% of patients had phantom limb pain, and six months later, 67% reported it. Some amputees experience continuous pain that varies in intensity or quality; others experience several bouts of pain per day, or it may reoccur less often. It is often described as shooting, crushing, burning or cramping. If the pain is continuous for a long period, parts of the intact body may become sensitized, so that touching them evokes pain in the phantom limb. Phantom limb pain may accompany urination or defecation.
   Section::::Signs and symptoms.
  
  A primary spontaneous pneumothorax (PSP) tends to occur in a young adult without underlying lung problems, and usually causes limited symptoms. Chest pain and sometimes mild breathlessness are the usual predominant presenting features. People who are affected by a PSP are often unaware of the potential danger and may wait several days before seeking medical attention. PSPs more commonly occur during changes in atmospheric pressure, explaining to some extent why episodes of pneumothorax may happen in clusters. It is rare for a PSP to cause a tension pneumothorax.
   Section::::Prognosis.
   BULLET::::- Tinel's sign is a classic test to detect median nerve irritation. Tinel's sign is performed by lightly tapping the skin over the flexor retinaculum to elicit a sensation of tingling or "pins and needles" in the median nerve distribution. Tinel's sign (pain or paresthesias of the median-innervated fingers with percussion over the median nerve), depending on the study, has 38–100% sensitivity and 55–100% specificity for the diagnosis of CTS.
  
  BULLET::::- Durkan test, "carpal compression test", or applying firm pressure to the palm over the nerve for up to 30 seconds to elicit symptoms has also been proposed.
   As with myocardial ischaemia referred pain in a certain portion of the body can lead to a diagnosis of the correct local center. Somatic mapping of referred pain and the corresponding local centers has led to various topographic maps being produced to aid in pinpointing the location of pain based on the referred areas. For example, local pain stimulated in the esophagus is capable of producing referred pain in the upper abdomen, the oblique muscles, and the throat. Local pain in the prostate can radiate referred pain to the abdomen, lower back, and calf muscles. Kidney stones can cause visceral pain in the ureter as the stone is slowly passed into the excretory system. This can cause immense referred pain in the lower abdominal wall.
   Section::::Diagnosis.:Classification.
  
  BULLET::::- Type I, formerly known as reflex sympathetic dystrophy (RSD), Sudeck's atrophy, or algoneurodystrophy, does not exhibit demonstrable nerve lesions. As the vast majority of patients diagnosed with CRPS have this type, it is most commonly referred to in medical literature as Type I.
  
  BULLET::::- Type II, formerly known as causalgia, has evidence of obvious nerve damage. Despite there being evidence of nerve injury, the cause of the mechanisms of CRPS Type II are as unknown as the mechanisms of Type I.
   It is proposed that inflammation and alteration of pain perception in the central nervous system play important roles. It has been suggested that persistent pain and the perception of non-painful stimuli as painful may be caused by inflammatory molecules (IL-1, IL2, TNF-alpha) and neuropeptides (substance P) released from peripheral nerves. This release may be caused by inappropriate crosstalk between sensory and motor fibers at the affected site. CRPS is not a psychological illness, yet pain can cause psychological problems, such as anxiety and depression. There is often impaired social and occupational function.
   Characteristic symptoms include a sharp stabbing pains in the chest. These typically get worse with breathing in and occur within a small area. Spells of pain usually last less than a few minutes. Typically it begins at rest and other symptoms are absent. Concerns about the condition may result in anxiety.
  
  Section::::Causes.
  
  The underlying cause is unclear. Some believe the pain may be from the chest wall or irritation of an intercostal nerve. Risk factors include psychological stress. The pain is not due to the heart.
  
  Section::::Treatment.
   Hyperexcitability hypothesizes that referred pain has no central mechanism. However, it does say that there is one central characteristic that predominates. Experiments involving noxious stimuli and recordings from the dorsal horn of animals revealed that referred pain sensations began minutes after muscle stimulation. Pain was felt in a receptive field that was some distance away from the original receptive field. According to hyperexcitability, new receptive fields are created as a result of the opening of latent convergent afferent fibers in the dorsal horn. This signal could then be perceived as referred pain.
  