[Pulmonary edemas. Classification and mechanisms].
Water exchanges in the lung under physiological conditions, are still badly known. Starling's classical equation is a too simplified view of the phenomenon. In addition, a certain number of its factors are badly determined. Such is the case with the hydrostatic pressure and the oncotic pressure of the interstitium. In pathological conditions secondary edema is opposed to lesions of the alveolocapillary membrane and hemodynamic edema where the increase in intravascular pressure increases a loss of liquid between the junctions of the endothelial cells. Between these two situations there are edemas, of which the mechanism is still subject to discussion, for example altitude edema, edema in heroin addicts, neurogenic edema.
[Pulmonary edemas. Classification and mechanisms]. Water exchanges in the lung under physiological conditions, are still badly known. Starling's classical equation is a too simplified view of the phenomenon. In addition, a certain number of its factors are badly determined. Such is the case with the hydrostatic pressure and the oncotic pressure of the interstitium. In pathological conditions secondary edema is opposed to lesions of the alveolocapillary membrane and hemodynamic edema where the increase in intravascular pressure increases a loss of liquid between the junctions of the endothelial cells. Between these two situations there are edemas, of which the mechanism is still subject to discussion, for example altitude edema, edema in heroin addicts, neurogenic edema.